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Results 51-60 of 107 (Search time: 0.002 seconds).
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PreviewIssue DateTitleAuthor(s)
2012Bioengineered microbes in disease therapyPaton, A.; Morona, R.; Paton, J.
2018Streptococcus pneumoniae: transmission, colonization and invasionWeiser, J.; Ferreira, D.; Paton, J.
2010Central role of manganese in regulation of stress responses, physiology, and metabolism in Streptococcus pneumoniaeOgunniyi, A.; Mahdi, L.; Jennings, M.; McEwan, A.; McDevitt, C.; Van der Hoek, M.; Bagley, C.; Hoffmann, P.; Gould, K.; Paton, J.
2012Role of lipid rafts and flagellin in invasion of colonic epithelial cells by Shiga-toxigenic Escherichia coli O113:H21Rogers, T.; Thorpe, C.; Paton, A.; Paton, J.; McCormick, B.A.
2013Inhibition of water absorption and selective damage to human colonic mucosa are induced by subtilase cytotoxin produced by Escherichia coli O113:H21Gerhardt, E.; Masso, M.; Paton, A.; Paton, J.; Zotta, E.; Ibarra, C.; McCormick, B.A.
2010Importance of CXC chemokine receptor 2 in alveolar neutrophil and exudate macrophage recruitment in response to pneumococcal lung infectionHerbold, W.; Maus, R.; Hahn, I.; Ding, N.; Srivastava, M.; Christman, J.; Mack, M.; Reutershan, J.; Briles, D.; Paton, J.; Winter, C.; Welte, T.; Maus, U.
2011In vivo leukocyte changes induced by Escherichia coli subtilase cytotoxinWang, H.; Paton, A.; McColl, S.; Paton, J.; Bliska, J.B.
2010The MerR/NmlR Family Transcription Factor of Streptococcus pneumoniae responds to carbonyl stress and modulates hydrogen peroxide productionPotter, A.; Kidd, S.; McEwan, A.; Paton, J.
2010Efficacy profiles of Daptomycin for treatment of Iinvasive and noninvasive pulmonary infections with treptococcus pneumoniaeHenken, S.; Bohling, J.; Martens-Lobenhoffer, J.; Paton, J.; Ogunniyi, A.; Briles, D.; Salisbury, V.; Wedekind, D.; Bode-Boger, S.; Welsh, T.; Bange, F.; Welte, T.; Maus, U.
2011Contribution of serotype and genetic background to virulence of serotype 3 and serogroup 11 pneumococcal isolatesMcAllister, L.; Ogunniyi, A.; Stroeher, U.; Leach, A.; Paton, J.; Camilli, A.