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PreviewIssue DateTitleAuthor(s)
2017NAD deficiency, congenital malformations, and niacin supplementationShi, H.; Enriquez, A.; Rapadas, M.; Martin, E.; Wang, R.; Moreau, J.; Lim, C.; Szot, J.; Ip, E.; Hughes, J.; Sugimoto, K.; Humphreys, D.; McInerney-Leo, A.; Leo, P.; Maghzal, G.; Halliday, J.; Smith, J.; Colley, A.; Mark, P.; Collins, F.; et al.
2008Interference with splicing of Presenilin transcripts has potent dominant negative effects on Presenilin activityNornes, S.; Newman, M.; Verdile, G.; Wells, S.; Stoick-Cooper, C.; Tucker, B.; Frederich-Sleptsova, I.; Martins, R.; Lardelli, M.
2007Contributions of pneumolysin, pneumococcal surface protein A (PspA), and PspC to pathogenicity of Streptococcus pneumoniae D39 in a mouse modelOgunniyi, A.; Le Messurier, K.; Graham, R.; Watt, J.; Briles, D.; Stroeher, U.; Paton, J.
2011A variable region within the genome of Streptococcus pneumoniae contributes to strain-strain variation in virulenceHarvey, R.; Stroeher, U.; Ogunniyi, A.; Smith-Vaughan, H.; Leach, A.; Paton, J.; Neyrolles, O.
2012Contribution of a genomic accessory region encoding a putative cellobiose phosphotransferase system to virulence of Streptococcus PneumoniaeMcAllister, L.; Ogunniyi, A.; Stroeher, U.; Paton, J.; Chakravortty, D.
2011Extracellular matrix formation enhances the ability of streptococcus pneumoniae to cause invasive diseaseTrappetti, C.; Ogunniyi, A.; Oggioni, M.; Paton, J.; Miyaji, E.N.
2018A Upf3b-mutant mouse model with behavioral and neurogenesis defectsHuang, L.; Shum, E.; Jones, S.; Lou, C.-H.; Dumdie, J.; Kim, H.; Roberts, A.; Jolly, L.; Espinoza, J.; Skarbrevik, D.; Phan, M.; Cook-Andersen, H.; Swerdlow, N.; Gecz, J.; Wilkinson, M.

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