1. Adelaide Research & Scholarship

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Results 1-10 of 11 (Search time: 0.002 seconds).
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PreviewIssue DateTitleAuthor(s)
1996A powerful new technique for isolating genes encoding cell surface antigens using retroviral expression cloningZannettino, A.; Rayner, J.; Ashman, L.; Gonda, T.; Simmons, P.
1995Activating point mutations in the common beta subunit of the human GM-CSF, IL-3 and IL-5 receptors suggest the involvement of beta subunit dimerization and cell type-specific molecules in signallingJenkins, B.; D'Andrea, R.; Gonda, T.
1996Homologous Regulation of the Rat C1a Calcitonin Receptor (CTR) in Nonosteoclastic Cells is Independent of CTR Messenger Ribonucleic Acid Changes and Cyclis Adenosine 3', 5' -Monophospate-Dependent Protein Kinase ActivationFindlay, D.; Houssami, S.; Christopoulos, G.; Sexton, P.
1996DNA-dependent protein kinase catalytic subunit: a target for an ICE-like protease in apoptosisSong, Q.; Lees-Miller, S.; Kumar, S.; Zhang, N.; Chan, D.; Smith, G.; Jackson, S.; Alnemri, E.; Litwack, G.; Khanna, K.; Lavin, M.
1999Calcitonin Receptor-Mediated Growth Suppression of HEK-293 Cells is Accompanied by Induction of p21 WAF1/CIP1 and G2/M ArrestEvdokiou, A.; Raggatt, L.; Findlay, D.; Atkins, G.
1995Inhibition of apoptosis by the expression of antisense Nedd2Kumar, S.
1999A cell type-specific constitutive point mutant of the common β-subunit of the human granulocyte-macrophage colony-stimulating factor (GM-CSF), interleukin (IL)-3, and IL-5 receptors requires the GM-CSF receptor α-subunit for activationJenkins, B.; Le, F.; Gonda, T.
1998Caspase-mediated cleavage of the ubiquitin-protein ligase Nedd4 during apoptosisHarvey, K.; Harvey, N.; Michael, J.; Parasivam, G.; Waterhouse, N.; Alnemri, E.; Watters, D.; Kumar, S.
1996A novel activating anti-β1 integrin monoclonal antibody binds to the cysteine-rich repeats in the β1 chainFaull, R.; Wang, J.; Leavesley, D.; Puzon, W.; Russ, G.; Vestweber, D.; Takada, Y.
1996Regulation of osteopontin expression by type 1 collagen in preosteoblastic UMR201 cellsTraianedes, K.; Martin, T.; Findlay, D.

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