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Results 111-120 of 125 (Search time: 0.003 seconds).
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PreviewIssue DateTitleAuthor(s)
2014Genetic diversity and virulence potential of shiga toxin-producing Escherichia coli O113: H21 strains isolated from clinical, environmental, and food sourcesFeng, P.; Delannoy, S.; Lacher, D.; dos Santos, L.; Beutin, L.; Fach, P.; Rivas, M.; Hartland, E.; Paton, A.; Guth, B.; Björkroth, J.
2014The impact of pneumolysin on the macrophage response to Streptococcus pneumoniae is strain-dependentHarvey, R.; Hughes, C.; Paton, A.; Trappetti, C.; Paton, J.; Tweten, R.; Neyrolles, O.
2013The signal sequence influences post-translational ER translocation at distinct stagesJohnson, N.; Hassdenteufel, S.; Theis, M.; Paton, A.; Paton, J.; Zimmermann, R.; High, S.; Palazzo, A.F.
2014The BiP molecular chaperone plays multiple roles during the biogenesis of torsinA, an AAA⁺ ATPase associated with the neurological disease early-onset torsion dystoniaZacchi, L.; Wu, H.; Bell, S.; Millen, L.; Paton, A.; Paton, J.; Thomas, P.; Zolkiewski, M.; Brodsky, J.
2014Opposing unfolded-protein-response signals converge on death receptor 5 to control apoptosisLu, M.; Lawrence, D.; Marsters, S.; Acosta-Alvear, D.; Kimmig, P.; Mendez, A.; Paton, A.; Paton, J.; Walter, P.; Ashkenazi, A.
2011Measuring signaling by the unfolded protein responseCox, D.; Strudwick, N.; Ali, A.; Paton, A.; Paton, J.; Schroder, M.; Conn, P.
2017Chaperone-driven degradation of a misfolded proinsulin mutant in parallel with restoration of wild-type insulin secretionCunningham, C.; He, K.; Arunagiri, A.; Paton, A.; Paton, J.; Arvan, P.; Tsai, B.
1998Antibodies to lipopolysaccharide block adherence of shiga toxin-producing Escherichia coli to human intestinal epithelial (Henle 407) cells.Paton, A.; Voss, E.; Manning, P.; Paton, J.
2017GRP78 is an important host factor for Japanese encephalitis virus entry and replication in mammalian cellsNain, M.; Mukherjee, S.; Karmakar, S.; Paton, A.; Paton, J.; Abdin, M.; Basu, A.; Kalia, M.; Vrati, S.; Diamond, M.S.
2017Autoinducer 2 signaling via the phosphotransferase FruA drives galactose utilization by streptococcus pneumoniae, resulting in hypervirulenceTrappetti, C.; McAllister, L.; Chen, A.; Wang, H.; Paton, A.; Oggioni, M.; McDevitt, C.; Paton, J.; McDaniel, L.

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