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PreviewIssue DateTitleAuthor(s)
2003Hepatocyte turnover during resolution of a transient hepadnaviral infectionSummers, J.; Jilbert, A.; Yang, W.; Aldrich, C.; Saputelli, J.; Litwin, S.; Toll, E.; Mason, W.
2005Covalently closed circular DNA is the predominant form of duck hepatitis B virus DNA that persists following transient infectionLe Mire, M.; Miller, D.; Foster, W.; Burrell, C.; Jilbert, A.
1998Protective efficacy of DNA vaccines against duck hepatitis B virus infection.Triyatni, M.; Jilbert, A.; Qiao, M.; Miller, D.; Burrell, C.
2013Nucleic acid polymers prevent the establishment of duck hepatitis B virus infection in vivoNoordeen, F.; Vaillant, A.; Jilbert, A.
2010The development of persistent duck hepatitis B virus infection can be prevented using antiviral therapy combined with DNA or recombinant fowlpoxvirus vaccinesFeng, F.; Teoh, C.; Qiao, Q.; Boyle, D.; Jilbert, A.
2005Effect of antiviral treatment with entecavir on age- and dose-related outcomes of duck hepatitis B virus infectionFoster, W.; Miller, D.; Scougall, C.; Kotlarski, I.; Colonno, R.; Jilbert, A.
2004Studying host immune responses against duck Hepatitis B virus infectionMiller, D.; Bertram, E.; Scougall, C.; Kotlarski, I.; Jilbert, A.
2000Immune responses to duck hepatitis B virus infectionJilbert, A.; Kotlarski, I.
1998Characterization of age- and dose-related outcomes of duck hepatitis B virus infectionJilbert, A.; Botten, J.; Miller, D.; Bertram, E.; de la M. Hall, P.; Kotlarski, I.; Burrell, C.
1996Kinetics of duck hepatitis B virus infection following low dose virus inoculation: One virus DNA genome is infectious in neonatal ducksJilbert, A.; Miller, D.; Scougall, C.; Turnbull, H.; Burrell, C.

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