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Please use this identifier to cite or link to this item: https://hdl.handle.net/2440/38958
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dc.contributor.authorKendall, H.-
dc.contributor.authorMarshall, R.-
dc.contributor.authorBartold, P.-
dc.date.issued2001-
dc.identifier.citationOral Diseases, 2001; 7(1):2-10-
dc.identifier.issn1354-523X-
dc.identifier.issn1601-0825-
dc.identifier.urihttp://hdl.handle.net/2440/38958-
dc.descriptionCopyright © 2001 Munksgaard. All rights reserved. The definitive version is available at www.blackwell-synergy.com-
dc.description.abstractNitric oxide (NO) is a free radical which has complex roles in both health and disease. It is now recognized that NO is essential for a vast spectrum of intracellular and extracellular events in a wide variety of tissues. NO has also been implicated in the pathogenesis of numerous inflammatory and autoimmune diseases. In this review we consider the roles of NO generally and in particular the implications for periodontal diseases.-
dc.description.statementofresponsibilityHK Kendall, RI Marshall, PM Bartold-
dc.language.isoen-
dc.publisherBlackwell Munksgaard-
dc.subjectT-Lymphocytes, Helper-Inducer-
dc.subjectAnimals-
dc.subjectHumans-
dc.subjectPeriodontitis-
dc.subjectDNA Damage-
dc.subjectNitric Oxide-
dc.subjectFree Radical Scavengers-
dc.subjectSecond Messenger Systems-
dc.subjectNeutrophil Activation-
dc.subjectHomeostasis-
dc.titleNitric oxide and tissue destruction-
dc.typeJournal article-
dc.identifier.doi10.1034/j.1601-0825.2001.70102-
pubs.publication-statusPublished-
dc.identifier.orcidBartold, P. [0000-0002-5695-3877]-
Appears in Collections:Aurora harvest
Dentistry publications

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