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Issue Date
Title
Author(s)
2002
High frequency of point mutations clustered within the adenosine triphosphate-binding region of BCR/ABL in patients with chronic myeloid leukemia or Ph-positive acute lymphoblastic leukemia who develop imatinib (STI571) resistance
Branford, S.
;
Rudzki, Z.
;
Walsh, S.
;
Grigg, A.
;
Arthur, C.
;
Taylor, K.
;
Herrmann, R.
;
Lynch, K.
;
Hughes, T.
2005
Dynamics of chronic myeloid leukaemia
Michor, F.
;
Hughes, T.
;
Iwasa, Y.
;
Branford, S.
;
Shah, N.
;
Sawyers, C.
;
Nowak, M.
2009
Clinical strategies to achieve an early and successful response to tyrosine kinase inhibitor therapy
Hughes, T.
;
Hochhaus, A.
2004
Clinical resistance to imatinib: mechanisms and implications
Hochhaus, A.
;
Hughes, T.
2009
Impact of baseline BCR-ABL mutations on response to Nilotinib in patients with chronic myeloid leukemia in chronic phase
Hughes, T.
;
Saglio, G.
;
Branford, S.
;
Soverini, S.
;
Kim, J.
;
Muller, M.
;
Martinelli, G.
;
Cortes, J.
;
Beppu, L.
;
Gottardi, E.
;
Dongho, K.
;
Erben, P.
;
Shou, Y.
;
Haque, A.
;
Gallagher, N.
;
Radich, J.
;
Hochhaus, A.
2009
Selecting optimal second-line tyrosine kinase inhibitor therapy for chronic myeloid leukemia patients after imatinib failure: does the BCR-ABL mutation status really matter?
Branford, S.
;
Vaz de Melo, J.
;
Hughes, T.
2006
OCT-1-mediated influx is a key determinant of the intraceflular uptake of imatinib but not nilotinib, (AMN107): reduced OCT-1 activity is the cause of low in vitro sensitivity to imatinib
White, D.
;
Saunders, V.
;
Dang, P.
;
Engler, J.
;
Zannettino, A.
;
Cambareri, A.
;
Quinn, S.
;
Manley, P.
;
Hughes, T.
2007
Most CML patients who have a suboptimal response to imatinib have low OCT-1 activity: higher doses of imatinib may overcome the negative impact of low OCT-1 activity
White, D.
;
Saunders, V.
;
Dang, P.
;
Engler, J.
;
Venables, A.
;
Zrim, S.
;
Zannettino, A.
;
Lynch, K.
;
Manley, P.
;
Hughes, T.
2003
Detection of BCR-ABL mutations in patients with CML treated with imatinib is virtually always accompanied by clinical resistance, and mutations in the ATP phosphate-binding loop (P-loop) are associated with a poor prognosis
Branford, S.
;
Rudzki, Z.
;
Walsh, S.
;
Parkinson, I.
;
Grigg, A.
;
Szer, J.
;
Taylor, K.
;
Hermann, R.
;
Seymour, J.
;
Arthur, C.
;
Joske, D.
;
Lynch, K.
;
Hughes, T.
Discover
Author
5
Branford, S.
3
Hochhaus, A.
3
Lynch, K.
2
Arthur, C.
2
Dang, P.
2
Engler, J.
2
Grigg, A.
2
Manley, P.
2
Rudzki, Z.
2
Saunders, V.
.
next >
Subject
9
Benzamides
9
Humans
9
Imatinib Mesylate
9
Leukemia, Myelogenous, Chronic, B...
9
Pyrimidines
6
Fusion Proteins, bcr-abl
4
Mutation
4
Protein Kinase Inhibitors
3
Adult
3
Antineoplastic Agents
.
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Date issued
3
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2005
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2004
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2002