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Results 101-110 of 127 (Search time: 0.005 seconds).
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Issue Date
Title
Author(s)
2007
Imatinib increases the intracellular concentration of nilotinib which may explain the observed synergy between these drugs
White, D.
;
Saunders, V.
;
Quinn, S.
;
Manley, P.
;
Hughes, T.
2013
Prospective histomorphometric and DXA evaluation of bone remodeling in imatinib-treated CML patients: Evidence for site-specific skeletal effects
Vandyke, K.
;
Fitter, S.
;
Drew, J.
;
Fukumoto, S.
;
Schultz, C.
;
Sims, N.
;
Yeung, D.
;
Hughes, T.
;
Zannettino, A.
2003
Detection of BCR-ABL mutations in patients with CML treated with imatinib is virtually always accompanied by clinical resistance, and mutations in the ATP phosphate-binding loop (P-loop) are associated with a poor prognosis
Branford, S.
;
Rudzki, Z.
;
Walsh, S.
;
Parkinson, I.
;
Grigg, A.
;
Szer, J.
;
Taylor, K.
;
Hermann, R.
;
Seymour, J.
;
Arthur, C.
;
Joske, D.
;
Lynch, K.
;
Hughes, T.
2010
Phase III, randomized, open-label study of daily Imatinib Mesylate 400 mg versus 800 mg in patients with newly diagnosed, previously untreated chronic myeloid leukemia in chronic phase using molecular end points: Tyrosine Kinase Inhibitor Optimization and Selectivity Study
Cortes, A.
;
Baccarani, M.
;
Guilhot, F.
;
Druker, B.
;
Branford, S.
;
Kim, D.
;
Pane, F.
;
Pasquini, R.
;
Goldberg, S.
;
Kalaycio, M.
;
Moiraghi, B.
;
Rowe, J.
;
Tothova, E.
;
de Souza, C.
;
Rudoltz, M.
;
Yu, R.
;
Krahnke, T.
;
Kantarjian, H.
;
Radich, J.
;
Hughes, T.
2010
Functional activity of the OCT-1 protein is predictive of long-term outcome in patients with chronic-phase chronic myeloid leukemia treated with Imatinib
White, D.
;
Dang, P.
;
Engler, J.
;
Frede, A.
;
Osborn, M.
;
Saunders, V.
;
Manley, P.
;
Zrim, S.
;
Hughes, T.
2007
Measurement of in vivo BCR-ABL kinase inhibition to monitor imatinib-induced target blockade and predict response in chronic myeloid leukemia
White, D.
;
Saunders, V.
;
Grigg, A.
;
Arthur, C.
;
Filshie, R.
;
Leahy, M.
;
Lynch, K.
;
To, L.
;
Hughes, T.
2011
BCR-ABL transcript dynamics support the hypothesis that leukemic stem cells are reduced during imatinib teatment
Stein, A.
;
Bottino, D.
;
Modur, V.
;
Branford, S.
;
Kaeda, J.
;
Goldman, J.
;
Hughes, T.
;
Radich, J.
;
Hochhaus, A.
2011
SHP-1 expression accounts for resistance to imatinib treatment in Philadelphia chromosome-positive cells derived from patients with chronic myeloid leukemia
Esposito, N.
;
Colavita, I.
;
Quintarelli, C.
;
Sica, A.
;
Peluso, A.
;
Luciano, L.
;
Picardi, M.
;
Vecchio, L.
;
Buonomo, T.
;
Hughes, T.
;
White, D.
;
Radich, J.
;
Russo, D.
;
Branford, S.
;
Saglio, G.
;
Vaz de Melo, J.
;
Martinelli, R.
;
Ruoppolo, M.
;
Kalebic, T.
;
Martinelli, G.
;
et al.
2008
Dasatinib cellular uptake and efflux in chronic myeloid leukemia cells: therapeutic implications
Hiwase, D.
;
Saunders, V.
;
Hewett, D.
;
Frede, A.
;
Zrim, S.
;
Dang, P.
;
Eadie, L.
;
To, L.
;
Vaz de Melo, J.
;
Kumar, S.
;
Hughes, T.
;
White, D.
2013
Protein kinase activity of phosphoinositide 3-kinase regulates cytokine-dependent cell survival
Thomas, D.
;
Powell, J.
;
Green, B.
;
Barry, E.
;
Ma, Y.
;
Woodcock, J.
;
Fitter, S.
;
Zannettino, A.
;
Pitson, S.
;
Hughes, T.
;
Lopez, A.
;
Shepherd, P.
;
Wei, A.
;
Ekert, P.
;
Guthridge, M.
;
Hunter, T.
Discover
Author
37
White, D.
30
Branford, S.
24
Hochhaus, A.
14
Radich, J.
14
To, L.
13
Kantarjian, H.
13
Saglio, G.
13
Saunders, V.
13
Townsend, G.
13
Zannettino, A.
.
next >
Subject
74
Leukemia, Myelogenous, Chronic, B...
73
Pyrimidines
63
Imatinib Mesylate
61
Piperazines
60
Benzamides
46
Antineoplastic Agents
45
Female
44
Male
43
Fusion Proteins, bcr-abl
41
Protein Kinase Inhibitors
.
next >
Date issued
6
2020 - 2022
52
2010 - 2019
65
2000 - 2009
4
1995 - 1999