1. Adelaide Research & Scholarship

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Results 21-30 of 144 (Search time: 0.002 seconds).
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PreviewIssue DateTitleAuthor(s)
2004Inhibition of generation of cytotoxic T lymphocyte activity by a CCL19/macrophage inflammatory protein (MIP)-3beta antagonistPilkington, K.; Clark-Lewis, I.; McColl, S.
2003Defining the role for XAP2 in stabilization of the dioxin receptorLees, M.; Peet, D.; Whitelaw, M.
2006ZNF652, a novel zinc finger protein, interacts with the putative breast tumor suppressor CBFA2T3 to repress transcriptionKumar, R.; Manning, J.; Spendlove, H.; Kremmidiotis, G.; McKirdy, R.; Lee, J.; Millband, D.; Cheney, K.; Stampfer, M.; Dwivedi, P.; Morris, H.; Callen, D.
2002Differential activities of murine single minded 1 (SIM1) and SIM2 on a hypoxic response element - Cross-talk between basic helix-loop-helix/Per-Arnt-Sim homology transcription factorsWoods, S.; Whitelaw, M.
2007Lung-specific overexpression of CC chemokine ligand (CCL) 2 enhances the host Defense to Streptococcus pneumoniae infection in mice: Role of the CCL2-CCR2 axisWinter, C.; Taut, K.; Srivastava, M.; Langer, F.; Mack, M.; Briles, D.; Paton, J.; Maus, R.; Welte, T.; Gunn, M.; Maus, U.
2001The protein tyrosine phosphatase TCPTP suppresses the tumorigenicity of glioblastoma cells expressing a mutant epidermal growth factor receptorKlingler-Hoffmann, M.; Fodero-Tavoletti, M.; Mishima, K.; Narita, Y.; Cavenee, W.; Furnari, F.; Huang, H.; Tiganis, T.
2002Asparagine hydroxylation of the HIF transactivation domain: A hypoxic switchLando, D.; Peet, D.; Whelan, D.; Gorman, J.; Whitelaw, M.
2003RegR, a global LacI/GalR family regulator, modulates virulence and competence in Streptococcus pneumoniaeChapuy-Regaud, S.; Ogunniyi, A.; Diallo, N.; Huet, Y.; Desnottes, J.; Paton, J.; Escaich, S.; Trombe, M.
2009Chaperone-targeting cytotoxin and endoplasmic reticulum stress-inducing drug synergize to kill cancer cellsBacker, J.; Krivoshein, A.; Hamby, C.; Pizzonia, J.; Gilbert, K.; Ray, Y.; Brand, H.; Paton, A.; Paton, J.; Backer, M.
2005Activity of hypoxia-inducible factor 2α is regulated by association with the NF-κB essential modulatorBracken, C.; Whitelaw, M.; Peet, D.

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